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- Your help will ease human suffering and save lives -
Donation instructions: see below & Home Page
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We at STAF, Inc. want to know how this advice website has improved your & your family's life
Let us know in your donation letter
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When we lose weight, where does the lost weight go?
Important information - study well & apply
Quotation "Knowledge is no power - only applied knowledge is power"
(Dr. Christian, STAF, Inc.'s founding President)
Click green for further info
When we lose weight, where does the lost weight go? After weeks of trimming a few calories here, exercising a little more there, you put on a pair of jeans, and like magic, the waist has grown. You button them up to behold a welcome space between your belly and the fabric. How did that happen?
With 66 percent of the adult American population either overweight or obese [source: Centers for Disease Control and Prevention], a lot of people are trying to drop some pounds. Through dieting, exercise, surgery or a host of other alternatives, they hope to reach the goal of a smaller body. But to where does that weight disappear when the hard work pays off?
The short answer is that our bodies convert molecules in fat cells to usable forms of energy, thus shrinking the cells. But getting this to happen isn't just about sweat bands and short shorts. Understanding how our bodies perform this tummy-trimming trick requires a little more detail.
We know that weight loss hinges on burning calories. Calories measure the potential energy in food you eat in the form of fats, proteins and carbohydrates.
If our bodies were cars, energy would be the gas to keep everything running. Lounging in front of the television is like cruising the strip, while sprinting around a track is more like drag racing at maximum speeds. In short, more work means more energy.
The body uses some of those calories to digest food. Once the food is broken down into its respective parts of carbohydrates, fats and proteins, it either uses the remaining energy or converts it to fat for storage in fat cells. Fat cells live in adipose tissue, which basically acts like an internal gas station, storing away fuel reserves.
To lose weight, you must burn more calories, or energy, than you consume to start using up that fuel reserve. Essentially, you're not ingesting enough calories to fuel your additional exercise, so your body must pull from fat stores.
According to the Law of Conservation of Mass, matter is neither created nor destroyed, but it may alter its form through chemical reaction. Essentially, that tells us that while we lose mass in our bodies by burning up fat, it does not just disappear. It simply changes form, like water and steam.
When we eat, the glucose and sugar harnessed from carbohydrates are the first fuel sources. The liver stores the glucose in the form of glycogen and releases it into the bloodstream as necessary to keep our body trucking along. Think of your bloodstream as an interconnected conveyor belt that takes necessary nutrients to the body parts that need them. Once that glucose runs out, fat takes over. Harnessing energy by burning fat is referred to as ketosis.
Hormones regulating our blood sugar levels activate an enzyme in the blood vessels of fat tissue called lipase. Lipase ignites fat cells to release macromolecules called triglycerides, which are what make fat cells fat. Triglycerides are made of glycerol and three fatty acid chains. When they receive the signal from lipase to exit the fat cells, the triglycerides break up into their respective components and enter the bloodstream for use. The liver snatches up the glycerol to break it down for energy, and some of the fatty acids move to the muscles that can farm them for energy as well.
This action of breaking down triglycerides into usable energy is called lipolysis. Once inside the mitochrondia, or power source, or muscle or liver cells, the components of the glycerol and fatty acids are shuffled and reshuffled to harness their energy potential, producing heat, water, carbon dioxide and adenosine triphosphate (ATP). ATP hauls potential energy in its molecular bonds for use when we exercise like cellular carb loading. The water exits our bodies as sweat and urine, and we exhale the carbon dioxide.
Now that the body has relieved fat cells of some glycerol and fatty acids, they get smaller. But if the fat tissue shrinks, what happens to our skin?
CALORIE BREAKDOWN
1 g Carbohydrates: 4 calories
1 g Protein: 4 calories
1 g Fat: 9 calories
[source: click: How Calories Work]
Lost Weight = Loose Skin
If our jeans hang loose when we lose weight, it would make sense that our skin would as well. In cases of moderate weight loss, our skin shrinks back to fit the body's new size, thanks to its elasticity. A protein called collagen in the skin gives it this special property, which helps explain how we don't outgrow our skin as we get older. As we age, these collagen fibers in the skin weaken, leading to wrinkles.
Collagen does have its limits. Rapid growth or weight gain can outpace collagen production in the skin, causing areas to overstretch. This can lead to striations called stretch marks*). These are particularly common with pregnancy and adolescents going through puberty. Although a number of lotions and creams on the market claim to get rid of stretch marks, most naturally go away on their own. *) stretch marks = streaks or stripes on the skin, especially on the abdomen, caused by distension**) of the skin from obesity or during pregnancy; **) distension click: Abdominal distension
Sometimes in cases of massive weight loss, people have folds of extra skin left over from their heavier days. Their skin had become so outstretched that it hangs limply from the thinner body, like worn out elastic.
As the number of obese people undergoing gastric bypass surgery increases, so do corrective surgeries for the unwanted and often uncomfortable loose skin. Although it may seem purely cosmetic, extra skin can actually cause infections, rashes and back problems. Since it's a highly invasive procedure, plastic surgeons may space out skin tucks and removals over months or a year, costing up to $100,000.
Even after the work is done, patients wear lifelong scars from the surgeries. Also, this type of body recontouring comes with risks. Since it's a relatively new procedure, doctors only know anecdotal evidence of complications, including dead skin, infection and open wounds [source: Singer]. But as beltlines continue to bulge, the numbers of patients will likely increase as it has the past decade.
RELATED ARTICLES- click and study
- Body Mass Index
- How Fat Cells Work
- Is lack of sleep making me fat?
- American Society of Plastic Surgeons -- Statistics
- MedlinePlus -- Weight Control
Sources for the above article:
- MedlinePlus Medical Encyclopedia. "Striae." National Library of Medicine. Updated April 16, 2007. (April 25, 2008)
http://www.nlm.nih.gov/medlineplus/ency/article/003287.htm
Regalado, Antonio. "After Weight-Loss Surgery -- More Surgery." The Wall Street Journal. Oct. 19, 2004.
Singer, Natasha. "Newly Petite Skin That's XL." The New York Times. Aug. 3, 2006. (April 25, 2008)
Sporny, Lora A. "When you lose weight where does it go?" Scientific American. Dec. 18, 2006. (April 25, 2008)
http://www.sciam.com/article.cfm?id=when-you-lose-weight-wher
Click: Study Finds Nearly 29% of World Population Is Overweight
The obesity epidemic is global: 2.1 billion people, or about 29% of the world's population, were either overweight or obese in 2013, and nearly two out of three of the obese live in developing countries, according to click: a study released
______________
Seeking Clues to Obesity in Rare Hunger Disorder
The Prader-Willi Syndrome Association knows of at least 8,000 Americans with the condition
Most patients are missing a chunk of chromosome 15
Others have two complete copies of the chromosome
Lisa Tremblay still recalls in horror the time her daughter Kristin pulled a hot dog crawling with ants from the garbage at a cookout and prepared to swallow it.
Kristin has a rare genetic abnormality that gives her an incessant, uncontrollable hunger. Some people with the condition, called Prader-Willi syndrome, will eat until their stomach ruptures and they die. And, not surprisingly, many are obese. click: Prader–Willi syndrome
“She’s eaten dog food. She’s eaten cat food,” said Ms. Tremblay, who lives in Nokomis, Fla. When Kristin, now 28, was a child, neighbors once called social welfare authorities, thinking Kristin was not being fed because she complained of being hungry so much.
Once an obscure and neglected disease, Prader-Willi is starting to attract more attention from scientists and pharmaceutical companies for a simple reason: It may shed some light on the much broader public health problems of overeating and obesity.
“These are remarkable human models of severe obesity,” said Dr. Steven B. Heymsfield, a professor and former executive director of the Pennington Biomedical Research Center in Baton Rouge, La. “When we discover the underlying mechanism of these very rare disorders, they will shed light on garden-variety obesity.”
A typical dinner for Ms. Tremblay: salad, green beans, fruit, chocolate-covered cherries and Crystal Light for 450 calories.
One drug being developed to help obese people lose weight has shown some preliminary signs of success in patients with Prader-Willi. The drug, beloranib, is believed to work by reducing fat synthesis and increasing fat use. In a small trial,
it reduced weight and body fat and lowered the food-seeking urge, according to the drug’s developer, Zafgen.
click: Zafgen
“This is the first thing that has really been tried and been at all successful in individuals with Prader-Willi,” said Dr. Jennifer Miller, associate professor of pediatric endocrinology click: Endocrinology at the University of Florida and the lead investigator of the trial.
There are reasons to be cautious about the link between Prader-Willi and general obesity because the mechanisms behind both are not well understood and could be different.
But Prader-Willi patient advocates are actively encouraging the association in hopes that linking the syndrome to the broader problem will attract more academic and pharmaceutical industry research on their disease. “The more interest and research there is on it, the more it helps our kids,” said Janalee Heinemann, director of research and medical affairs at the Prader-Willi Syndrome Association. Click: Prader-Willi Syndrome Associationwww.pwsausa.org/
There are also reasons to be cautious about the Zafgen results. The trial involved only 17 people, and the main part of it lasted only four weeks. On some measures — including the trial’s primary one of weight reduction — the difference between the drug and a placebo was not statistically significant.
Still, Thomas E. Hughes, Zafgen’s chief executive, said the results were strong enough that the company would conduct a larger study aimed at getting beloranib approved to treat Prader-Willi. click: Beloranib
“It gives us a way to establish the benefit of our drug in what would arguably be the toughest of patients to treat,” he said.
Another company, Ferring Pharmaceuticals, click: Ferring Pharmaceuticals
plans to begin a study of its drug carbetocin soon click: Carbetocin
The drug works in the same manner as oxytocin click: Oxytocin sometimes called the love hormone because it promotes human bonding. A small French study suggested oxytocin might not only improve behavioral problems associated with the disease but also curb appetites.
Rhythm, a start-up company, click: Rhythm Pharmaceuticals also plans to test its experimental obesity drug, RM-493, for Prader-Willi click: Rhythm: MC4/RM-493
Arena Pharmaceuticals click: Arena Pharmaceuticals which sells the weight loss drug Belviq
click: BELVIQ® , says Prader-Willi is an area of interest. And some academic trials are underway as well, making for what advocates say is an unusual amount of activity.
“Our biggest problem in 2014 and 2015 is making sure to have enough families to get the studies done,” said Dr. Theresa Strong, chairwoman of the scientific advisory board of the Foundation for Prader-Willi Research
click: Foundation for Prader-Willi Research
The Prader-Willi Syndrome Association knows of at least 8,000 Americans with the condition. Most patients are missing a chunk of chromosome 15. Others have two complete copies of the chromosome.
If the insatiable appetite were not enough, people with Prader-Willi also have slow metabolisms, meaning they gain weight exceedingly easily. Most also have intellectual disability and autistic behaviors.
Keeping weight in check is a constant battle for parents and caregivers.
“We lock up our refrigerator. We lock up our freezer. We lock up the pantry,” said Mark Greenberg of Denver, a consultant to a hedge fund whose 14-year-old son Zachary has Prader-Willi.
The family even locks the garbage pail in the kitchen. Alarms sound if Zachary tries to leave the house.
Appetite is a huge problem because it makes it impossible for people to live independently or to hold a job.
Jim Kane of Baltimore, Md., said his daughter Kate, who has a high school diploma, has been fired from several jobs for taking food from other employees.
She has also been arrested for shoplifting food, he said, once at an airport for taking a couple of granola bars.
Scientists do not understand the mechanisms behind Prader-Willi, though the insatiable appetite stems from dysfunction of the hypothalamus, the part of the brain that controls hunger.
Researchers say that as with other diseases, including Alzheimer’s and high cholesterol, many things might be learned from studying the most extreme or earliest onset cases.
“These mechanisms, if we were able to understand them in Prader-Willi, would shed an incredible amount of light on appetite,” said Dr. Joan Han, an endocrinologist at the National Institutes of Health who is conducting research on the syndrome and other rare eating-related disorders.
Her own research, for instance, has found low levels of a protein called brain-derived neurotropic factor in the blood of people with Prader-Willi. Other studies have also linked lack of the protein to obesity.
But some experts say Prader-Willi patients differ from other obese people in ways like their high levels of the appetite-increasing hormone ghrelin.
“There are some distinctive characteristics of Prader-Willi syndrome that suggest it may not be a stand-in or model for all of obesity,” said Elisabeth M. Dykens, director of Vanderbilt University’s Vanderbilt Kennedy Center, which studies developmental disorders. click: Elisabeth Dykens, Ph.D. - Vanderbilt Kennedy Center
For Zafgen, however, Prader-Willi represents a potentially faster path to the market for a relatively limited investment. While beloranib has shown good results in early studies as a treatment for general obesity, winning approval for that use will probably require clinical trials involving thousands of patients to rule out serious side effects.
Winning approval for Prader-Willi might require testing in only dozens or hundreds of patients. And there might be more tolerance of side effects because there is a desperate need for treatments. Also, since Prader-Willi is a rare disease, Zafgen would qualify for tax breaks and some market exclusivity under the Orphan Drug Act and could potentially charge an extremely high price for the product. click: Orphan Drug Act - Food and Drug Administration
Zafgen’s beloranib works by inhibiting methionine aminopeptidase 2, a hormone click: METAP2
The inhibition appears to stimulate the burning of fat and reduce its formation.
The 17 patients in Zafgen’s study were residents of a home for people with Prader-Willi in Gainesville, Fla., where diet is strictly controlled. During the study, the daily calorie intake was increased 50 percent to make it easier to see if beloranib affected weight and appetite.
Kristin Tremblay was one of the participants in the study. Her mother said she did not notice big changes when Kristin came home right after the study ended.
Oddly, however, Kristin did have trouble finishing one salad. And she had not gained weight despite the increase in calories during the study.
“I would try it again,” Ms. Tremblay said. “It certainly didn’t hurt her.”
Source: (1) NYT topic text & idea, (2) STAF, Inc.
______________________________________________
The Prader-Willi Syndrome Association knows of at least 8,000 Americans with the condition
Most patients are missing a chunk of chromosome 15
Others have two complete copies of the chromosome
Lisa Tremblay still recalls in horror the time her daughter Kristin pulled a hot dog crawling with ants from the garbage at a cookout and prepared to swallow it.
Kristin has a rare genetic abnormality that gives her an incessant, uncontrollable hunger. Some people with the condition, called Prader-Willi syndrome, will eat until their stomach ruptures and they die. And, not surprisingly, many are obese. click: Prader–Willi syndrome
“She’s eaten dog food. She’s eaten cat food,” said Ms. Tremblay, who lives in Nokomis, Fla. When Kristin, now 28, was a child, neighbors once called social welfare authorities, thinking Kristin was not being fed because she complained of being hungry so much.
Once an obscure and neglected disease, Prader-Willi is starting to attract more attention from scientists and pharmaceutical companies for a simple reason: It may shed some light on the much broader public health problems of overeating and obesity.
“These are remarkable human models of severe obesity,” said Dr. Steven B. Heymsfield, a professor and former executive director of the Pennington Biomedical Research Center in Baton Rouge, La. “When we discover the underlying mechanism of these very rare disorders, they will shed light on garden-variety obesity.”
A typical dinner for Ms. Tremblay: salad, green beans, fruit, chocolate-covered cherries and Crystal Light for 450 calories.
One drug being developed to help obese people lose weight has shown some preliminary signs of success in patients with Prader-Willi. The drug, beloranib, is believed to work by reducing fat synthesis and increasing fat use. In a small trial,
it reduced weight and body fat and lowered the food-seeking urge, according to the drug’s developer, Zafgen.
click: Zafgen
“This is the first thing that has really been tried and been at all successful in individuals with Prader-Willi,” said Dr. Jennifer Miller, associate professor of pediatric endocrinology click: Endocrinology at the University of Florida and the lead investigator of the trial.
There are reasons to be cautious about the link between Prader-Willi and general obesity because the mechanisms behind both are not well understood and could be different.
But Prader-Willi patient advocates are actively encouraging the association in hopes that linking the syndrome to the broader problem will attract more academic and pharmaceutical industry research on their disease. “The more interest and research there is on it, the more it helps our kids,” said Janalee Heinemann, director of research and medical affairs at the Prader-Willi Syndrome Association. Click: Prader-Willi Syndrome Associationwww.pwsausa.org/
There are also reasons to be cautious about the Zafgen results. The trial involved only 17 people, and the main part of it lasted only four weeks. On some measures — including the trial’s primary one of weight reduction — the difference between the drug and a placebo was not statistically significant.
Still, Thomas E. Hughes, Zafgen’s chief executive, said the results were strong enough that the company would conduct a larger study aimed at getting beloranib approved to treat Prader-Willi. click: Beloranib
“It gives us a way to establish the benefit of our drug in what would arguably be the toughest of patients to treat,” he said.
Another company, Ferring Pharmaceuticals, click: Ferring Pharmaceuticals
plans to begin a study of its drug carbetocin soon click: Carbetocin
The drug works in the same manner as oxytocin click: Oxytocin sometimes called the love hormone because it promotes human bonding. A small French study suggested oxytocin might not only improve behavioral problems associated with the disease but also curb appetites.
Rhythm, a start-up company, click: Rhythm Pharmaceuticals also plans to test its experimental obesity drug, RM-493, for Prader-Willi click: Rhythm: MC4/RM-493
Arena Pharmaceuticals click: Arena Pharmaceuticals which sells the weight loss drug Belviq
click: BELVIQ® , says Prader-Willi is an area of interest. And some academic trials are underway as well, making for what advocates say is an unusual amount of activity.
“Our biggest problem in 2014 and 2015 is making sure to have enough families to get the studies done,” said Dr. Theresa Strong, chairwoman of the scientific advisory board of the Foundation for Prader-Willi Research
click: Foundation for Prader-Willi Research
The Prader-Willi Syndrome Association knows of at least 8,000 Americans with the condition. Most patients are missing a chunk of chromosome 15. Others have two complete copies of the chromosome.
If the insatiable appetite were not enough, people with Prader-Willi also have slow metabolisms, meaning they gain weight exceedingly easily. Most also have intellectual disability and autistic behaviors.
Keeping weight in check is a constant battle for parents and caregivers.
“We lock up our refrigerator. We lock up our freezer. We lock up the pantry,” said Mark Greenberg of Denver, a consultant to a hedge fund whose 14-year-old son Zachary has Prader-Willi.
The family even locks the garbage pail in the kitchen. Alarms sound if Zachary tries to leave the house.
Appetite is a huge problem because it makes it impossible for people to live independently or to hold a job.
Jim Kane of Baltimore, Md., said his daughter Kate, who has a high school diploma, has been fired from several jobs for taking food from other employees.
She has also been arrested for shoplifting food, he said, once at an airport for taking a couple of granola bars.
Scientists do not understand the mechanisms behind Prader-Willi, though the insatiable appetite stems from dysfunction of the hypothalamus, the part of the brain that controls hunger.
Researchers say that as with other diseases, including Alzheimer’s and high cholesterol, many things might be learned from studying the most extreme or earliest onset cases.
“These mechanisms, if we were able to understand them in Prader-Willi, would shed an incredible amount of light on appetite,” said Dr. Joan Han, an endocrinologist at the National Institutes of Health who is conducting research on the syndrome and other rare eating-related disorders.
Her own research, for instance, has found low levels of a protein called brain-derived neurotropic factor in the blood of people with Prader-Willi. Other studies have also linked lack of the protein to obesity.
But some experts say Prader-Willi patients differ from other obese people in ways like their high levels of the appetite-increasing hormone ghrelin.
“There are some distinctive characteristics of Prader-Willi syndrome that suggest it may not be a stand-in or model for all of obesity,” said Elisabeth M. Dykens, director of Vanderbilt University’s Vanderbilt Kennedy Center, which studies developmental disorders. click: Elisabeth Dykens, Ph.D. - Vanderbilt Kennedy Center
For Zafgen, however, Prader-Willi represents a potentially faster path to the market for a relatively limited investment. While beloranib has shown good results in early studies as a treatment for general obesity, winning approval for that use will probably require clinical trials involving thousands of patients to rule out serious side effects.
Winning approval for Prader-Willi might require testing in only dozens or hundreds of patients. And there might be more tolerance of side effects because there is a desperate need for treatments. Also, since Prader-Willi is a rare disease, Zafgen would qualify for tax breaks and some market exclusivity under the Orphan Drug Act and could potentially charge an extremely high price for the product. click: Orphan Drug Act - Food and Drug Administration
Zafgen’s beloranib works by inhibiting methionine aminopeptidase 2, a hormone click: METAP2
The inhibition appears to stimulate the burning of fat and reduce its formation.
The 17 patients in Zafgen’s study were residents of a home for people with Prader-Willi in Gainesville, Fla., where diet is strictly controlled. During the study, the daily calorie intake was increased 50 percent to make it easier to see if beloranib affected weight and appetite.
Kristin Tremblay was one of the participants in the study. Her mother said she did not notice big changes when Kristin came home right after the study ended.
Oddly, however, Kristin did have trouble finishing one salad. And she had not gained weight despite the increase in calories during the study.
“I would try it again,” Ms. Tremblay said. “It certainly didn’t hurt her.”
Source: (1) NYT topic text & idea, (2) STAF, Inc.
______________________________________________
Below an article from England - It is a humorous,
yet a serious article relating to using hypnosis to quit smoking
Hypnosis provided by a competent hypnotherapist will bring fast results
STAF, Inc. has leading Advanced Clinical Hypnotherapists to help you to clear your life challenges
STAF, Inc. provides high-level, nationwide & internationally certified continuing education training for professional Clinical Hypnotherapists and provides training for the most demanding Master Hypnotist level
Training for the newcomers to the Clinical Hypnotherapy field is also provided by STAF, Inc.
Clinical Hypnotherapy services are in a growing demand worldwide
For U.S. & worldwide training information contact STAF, Inc. via an email
I Was Too Clever to Quit Smoking
The desired results were achieved by this client
By SAM BYERS
One central paradox of the smoker’s life is that, despite swallowing an almost comically flagrant deception, most smokers like to regard themselves as pretty smart people. Step outside any bar or restaurant, nip around the back of a conference room at coffee time, and you’ll see them: a bitter huddle of smarter-than-thou smokers, shooting down society’s sacred cows. Want to know how many people died while exercising last year? Ask a smoker. Think smoking kills you? Think again. Every smoker you meet will tell you about their Aunt Jenny or Uncle Steve who sucked back 90 a day and lived to be 100, and even then died only because their houses fell on them. No delusion or hypocrisy escapes the smoker’s gaze. Except, of course, his own.
I say this with some authority, because I was one. From the age of 17 to 32, despite herculean effort, I just couldn’t smoke enough to feel smoked out. The smoker in me, apparently the brains of the operation, sneered at my every self-improving effort.
After I made several pitiable attempts to break free, it became clear that in order to give up smoking, I was going to have to give up being such a smart aleck. And what better way of achieving this than to give in to something I believed I was too smart for? The solution was obvious: I would humble myself through hypnosis.
My session with Stella Knight (the name she uses professionally, she told me) took place at her home in Norfolk, England, on a white sofa in her bungalow’s front room. To the burbling accompaniment of a CD called “Celestial Sanctity,” Stella started in with her patter.
“People think you have to be stupid to be hypnotized,” she said, “but it’s actually the opposite. The best people to hypnotize are the most intelligent ones. You’re going to be easy.”
Aha! I thought. She’s at it already! Look how she boosted my self-esteem a little bit there. Very clever. But look how I spotted it too. Look how astute I am.
Clearly, Stella had never encountered someone like me: so intelligent, so attuned to her every hypnotic deception, that he became as difficult to hypnotize as a stupid person.
“Now close your eyes,” Stella said, “and imagine yourself eating a plate of human hair.”
Then the music changed to something trancy. She told me to count backward from 10. For some reason, I became terrified that she was taking her clothes off while I wasn’t looking. Was that the whisper of her jogging bottoms I could hear? I imagined myself coming to on the shag-pile carpet, a nude, beaming Stella beside me, sparking up a cigarette.
“All done,” Stella said after what seemed like a few seconds (but was actually 20 minutes). I opened my eyes to see her sitting in an armchair opposite me, sparking up a cigarette.
“Why are you lighting a cigarette?” I said, not quite masking the panic in my voice.
“So you can say goodbye to it,” Stella said.
What a load of bunk, I thought as I stood outside her bungalow, 80 pounds (about $125) poorer and seemingly no less cocksure.
I had a packet of tobacco in my pocket (I had, of course, smoked heavily on the way over). I decided to take it out and think about having a cigarette, noting carefully my emotional response.
The coughing fit that followed was both rapid in onset and utterly debilitating in effect. Within seconds, I was doubled over, leaning on Stella’s garden gnome for support, tears pouring down my face, cords of mucus unspooling from my nostrils and a noise rising up from my lungs that, even as I was making it, disgusted me. This led to further gagging and wheezing, until ultimately, I ended up on my knees in supplication to the gnome, holding up what I hoped was a calming hand to the young mother across the street who, upon seeing me, warily swept her toddler into her arms.
The fit lasted at least five excruciating minutes. At one point I realized my hand was in my mouth, trying to extract imaginary human hair from my windpipe. By the end I was exhausted and completely unable to imagine smoking for fear of triggering another bout of respiratory violence.
In the weeks that followed, this happened again, always when I ordinarily would have smoked. I took these episodes for what they clearly were: welcome reminders of my boundless susceptibility to suggestion, which, as any good hypnotist will tell you, is just another term for intelligence.
Sam Byers is the author of “Idiopathy,” a novel.
Source: NYT Magazine
____________________________________________
yet a serious article relating to using hypnosis to quit smoking
Hypnosis provided by a competent hypnotherapist will bring fast results
STAF, Inc. has leading Advanced Clinical Hypnotherapists to help you to clear your life challenges
STAF, Inc. provides high-level, nationwide & internationally certified continuing education training for professional Clinical Hypnotherapists and provides training for the most demanding Master Hypnotist level
Training for the newcomers to the Clinical Hypnotherapy field is also provided by STAF, Inc.
Clinical Hypnotherapy services are in a growing demand worldwide
For U.S. & worldwide training information contact STAF, Inc. via an email
- Click: Doctor of Clinical Hypnotherapy - Wikipedia
- The Doctor of Clinical Hypnotherapy (DCH) degree is an unaccredited degree in the United States [1]. However, it is important to understand voluntary .The Doctor of Clinical Hypnotherapy (DCH) degree is an unaccredited degree in the United States [1]. However, it is important to understand voluntary ...American Institute of ... - Consumer Concerns - Hypnotherapy Training
I Was Too Clever to Quit Smoking
The desired results were achieved by this client
By SAM BYERS
One central paradox of the smoker’s life is that, despite swallowing an almost comically flagrant deception, most smokers like to regard themselves as pretty smart people. Step outside any bar or restaurant, nip around the back of a conference room at coffee time, and you’ll see them: a bitter huddle of smarter-than-thou smokers, shooting down society’s sacred cows. Want to know how many people died while exercising last year? Ask a smoker. Think smoking kills you? Think again. Every smoker you meet will tell you about their Aunt Jenny or Uncle Steve who sucked back 90 a day and lived to be 100, and even then died only because their houses fell on them. No delusion or hypocrisy escapes the smoker’s gaze. Except, of course, his own.
I say this with some authority, because I was one. From the age of 17 to 32, despite herculean effort, I just couldn’t smoke enough to feel smoked out. The smoker in me, apparently the brains of the operation, sneered at my every self-improving effort.
After I made several pitiable attempts to break free, it became clear that in order to give up smoking, I was going to have to give up being such a smart aleck. And what better way of achieving this than to give in to something I believed I was too smart for? The solution was obvious: I would humble myself through hypnosis.
My session with Stella Knight (the name she uses professionally, she told me) took place at her home in Norfolk, England, on a white sofa in her bungalow’s front room. To the burbling accompaniment of a CD called “Celestial Sanctity,” Stella started in with her patter.
“People think you have to be stupid to be hypnotized,” she said, “but it’s actually the opposite. The best people to hypnotize are the most intelligent ones. You’re going to be easy.”
Aha! I thought. She’s at it already! Look how she boosted my self-esteem a little bit there. Very clever. But look how I spotted it too. Look how astute I am.
Clearly, Stella had never encountered someone like me: so intelligent, so attuned to her every hypnotic deception, that he became as difficult to hypnotize as a stupid person.
“Now close your eyes,” Stella said, “and imagine yourself eating a plate of human hair.”
Then the music changed to something trancy. She told me to count backward from 10. For some reason, I became terrified that she was taking her clothes off while I wasn’t looking. Was that the whisper of her jogging bottoms I could hear? I imagined myself coming to on the shag-pile carpet, a nude, beaming Stella beside me, sparking up a cigarette.
“All done,” Stella said after what seemed like a few seconds (but was actually 20 minutes). I opened my eyes to see her sitting in an armchair opposite me, sparking up a cigarette.
“Why are you lighting a cigarette?” I said, not quite masking the panic in my voice.
“So you can say goodbye to it,” Stella said.
What a load of bunk, I thought as I stood outside her bungalow, 80 pounds (about $125) poorer and seemingly no less cocksure.
I had a packet of tobacco in my pocket (I had, of course, smoked heavily on the way over). I decided to take it out and think about having a cigarette, noting carefully my emotional response.
The coughing fit that followed was both rapid in onset and utterly debilitating in effect. Within seconds, I was doubled over, leaning on Stella’s garden gnome for support, tears pouring down my face, cords of mucus unspooling from my nostrils and a noise rising up from my lungs that, even as I was making it, disgusted me. This led to further gagging and wheezing, until ultimately, I ended up on my knees in supplication to the gnome, holding up what I hoped was a calming hand to the young mother across the street who, upon seeing me, warily swept her toddler into her arms.
The fit lasted at least five excruciating minutes. At one point I realized my hand was in my mouth, trying to extract imaginary human hair from my windpipe. By the end I was exhausted and completely unable to imagine smoking for fear of triggering another bout of respiratory violence.
In the weeks that followed, this happened again, always when I ordinarily would have smoked. I took these episodes for what they clearly were: welcome reminders of my boundless susceptibility to suggestion, which, as any good hypnotist will tell you, is just another term for intelligence.
Sam Byers is the author of “Idiopathy,” a novel.
Source: NYT Magazine
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